Reduced lung uptake of Iodine-123 metaiodobenzylguanidine in high-altitude pulmonary oedema
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چکیده
منابع مشابه
Reduced myocardial 123-iodine metaiodobenzylguanidine uptake: a prognostic marker in familial amyloid polyneuropathy.
BACKGROUND Transthyretin familial amyloid polyneuropathy is a hereditary form of amyloidosis characterized by sensorimotor and autonomic neuropathy, cardiac conduction defects, and infiltrative cardiomyopathy. Previous studies have suggested that myocardial sympathetic denervation assessed by 123-iodine metaiodobenzylguanidine (MIBG) imaging occurs early in disease progression. However, its pro...
متن کاملHigh altitude-induced pulmonary oedema.
Almost one mountain trekker or climber out of two develops several symptoms of high altitude illness after a rapid ascent (> 300 m/day) to an altitude above 4000 m. Individual susceptibility is the most important determinant for the occurrence of high altitude pulmonary oedema (HAPE). Symptoms associated with HAPE are incapacitating fatigue, chest tightness, dyspnoea at the slightest effort, or...
متن کاملHaemodynamic studies in high altitude pulmonary oedema.
The clinical and pathological features of acute pulmonary oedema of high altitudes have been the subject of several recent reports (Houston, 1960; Hultgren et al., 1961; Penialoza, 1962; Arias-Stella and Kruger, 1963; Nayak, Roy, and Narayanan, 1964; Menon, 1965; Singh et al., 1965), but the haemodynamic effects of the illness are not well delineated (Fred et al., 1962; Hultgren et al., 1964). ...
متن کاملHigh-altitude pulmonary oedema in native highlanders.
To cite: Yanamandra U, Patyal S, Mukherji R, et al. BMJ Case Rep Published online: [please include Day Month Year] doi:10.1136/ bcr-2013-202513 DESCRIPTION A 28-year-old Ladakhi police personnel, native highlander, serving at high altitude (∼12 000 ft) presented with a short history of cough and breathlessness while on leave for 10 days at the same altitude. There was no history of deinduction ...
متن کاملPathogenesis of high-altitude pulmonary oedema.
We have read with great interest the paper by WEST et al. [1], regarding the stress failure mechanism of pulmonary capillaries in the pathogenesis of high-altitude pulmonary oedema (HAPE), and would like to add some considerations. HACKETT et al. [2] described a high incidence of HAPE in subjects without the right pulmonary artery. Recently, we have reported a case of unilateral, left sided HAP...
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ژورنال
عنوان ژورنال: Respirology
سال: 2008
ISSN: 1323-7799,1440-1843
DOI: 10.1111/j.1440-1843.2008.01343.x